In Australia gout is a growing health problem.1 As healthcare practitioners we know that gout is caused from the deposition and accumulation of monosodium urate monohydrate crystals within joints.2 Increased serum uric acid (SUA) is considered a causal factor in gout patients and treatment considerations in both allopathic and complementary medicine focus on the reduction of uric acid (UA) in the gout patient. However, rising levels of UA also provide clues to early disease onset or warning signs for systemic inflammatory conditions such as obesity, hypertension, abnormal lipid metabolism, impaired glucose tolerance and chronic kidney disease.2 A bidirectional relationship of SUA, gout and metabolic syndrome exists and is therefore worth understanding.